Telogen effluvium (TE) is probably the second most common form of hair loss in dermatologists. It is a poorly defined condition; Very little research has been done to understand TE. Essentially, TE occurs when there is a change in the growing hairs of the hair follicles. The number of hair-growing hair follicles will show a significant increase in still, telogen-stage hair follicles, which will be significantly reduced for any reason during the rest or telogen phase. The result is spillage or hair loss.
TE may be confused as a common hair loss in hair, this hair may not fall out. In some parts of the head skin, it may be a bit more severe than the others. Often, the hair on the top of the scalp is thinner than the hair on the sides and behind the scalp. Except for a few rare chronic cases, there is usually no hair line recession.
The spilled hairs are typically telogenous fur, which can be recognized by a small keratin lamp at the root tip. Keratinized pellets do not matter whether they are pigmented or colorless; hair strands are still typical telogen hairs.
People with TE never lose all of their scalp hair, but in severe cases they may noticeably thin their hair. While TE is usually limited to the scalp, in more serious cases it may also affect other areas, such as the TE, eyebrows, or groin area.
Regardless of the shape of TE’s hair loss, it can be completely reversed. Hair follicles are not affected permanently or irreversibly; there is only more hair follicles in case of a rest that should normally be.e United States.
Treatments for Telogen Effluvium
How TE is treated depends on what drives it. For a short-term TE that can be connected to a trigger, such as surgery, the best response is to sit tight and wait for the follicles to recover spontaneously.
If the causal factor can be isolated for permanent TE, then the best method is to remove it. For example, if stress is a problem, stress reduction is a long-term answer. If a diet lacks in a blood test, reinforcements can work. The deficiency in thyroid hormones can be treated with hormone supplements.
However, a particular causal factor can not usually be identified. In this case, treatment options are low. Most dermatologists resort to prescribing minoxidil, which is a direct hair growth stimulant. Minoxidil can work well for some individuals with TE, but if the underlying cause is still present, it should be continued to prevent the re-development of minoxidil TE. Removal of the tetanus may stop the use of minoxidil.
Before leaving the topic of TE, there are a few words about natural hair loss. Everyone is shedding hair and you can see hair loss at certain times of the year. Research has shown that at least in Northern Europe, people who are far from ecovord, have fallen more hair in the fall and fewer hair in the spring.
The number of telogen hair follicles and this transient increase in hair loss are probably due to hormone changes in response to changes in daylight exposure. Studies in mink and other mammals show that exposure to daylight significantly changes prolactin levels and prolactin has a significant effect on molting. As with mink and other mammals, people probably have the same fermentation reaction. Hair loss should be temporary.
There are three basic ways that TE can develop.
1. It can be an environmental insult that makes “shocking” the growing hair follicles and decides to take a rest for a while. Hair loss and hair loss causes an increase in hair loss. This TE form can develop rapidly and can be noticed in a month or two after taking the shock. If the trigger is short-lived, the hair will return to its growing condition and begin to produce new hair strands quickly. This TE form usually lasts less than six months and the affected individual has a normal density of scalp again within a year.
2. The second form of TE develops slower and lasts longer. Hair follicles may not suddenly turn hairpins and become telogen in rest. On the contrary, the follicle may take a resting state as normal, but after a month or two, a new anagen does not return to the hair growth state but remains in telogen state for a long time.
This leads to gradual accumulation of hair follicles in the telogen, and an ever decreasing number of anagen hair roots leaving the growing hair. Hair loss in this TE form may not be noticed very much, but it will slowly thin the hair in the hair. This form of TE is more likely to occur in response to persistent triggering factors.
3. In a third type of TE, hair follicles do not remain stationary, but cycle through shortened growth cycles. When this happens, the individual continues to pour thin hair of the hair and short, thin hair of the hair.
Telogen Effluvium Reasons: Stress and Diet
What are the triggering factors for TE? The short answer is many and varied. Classical short-term TE usually happens to women after birth. The sudden change in hormone level at birth, called postpartum alopecia, shuts down for a period of time for the hair follicles. Hair loss can be a significant amount, but most women climb their hair quickly.
Similarly, physical trauma, such as grafts, collision diets, car bumps, and the possibility of surgery can sometimes be a shock to the system, and some of the hair follicles in the head go into winter sleep. As the environmental insult goes on and the body heals, TE decreases and new hair grows.
Some medicines can also induce TE, especially antidepressants. Often switching to a different drug solves the problem.
More persistent insults can lead to more permanent TE. For example, a chronic disease can cause TE. Probably the two most common problems are chronic stress and lack of diet. Many dermatologists believe that chronic stress can make a gradual negative impact on hair growth and lead to permanent TE. Investigations with animal models have provided evidence to support this claim. There seems to be a link between stress and hair follicles entering a state of telogen rest, and a change in hair follicle biochemistry.
Whether dietary problems are causing TE in North America is being discussed among dermatologists. Missing minerals, vitamins or essential amino acids can certainly cause TE, as diets in third world countries may be completely missing in one or more nutrients. Animal experiments also provide supportive evidence.
In the first world countries, the average diet is seldom deficient in a particular vitamin or mineral. However, some dermatologists argue that a reduction in red meat consumption and a vegetarian dietary preference and that not all the nutrients necessary for some people’s general growth of good hair and body are balanced. In particular, there are claims that women are inadequate in iron intake. Why women in private? Because women lose menstruation periodically at regular intervals.
While some dermatologists now consume less red meat, an important source of iron, some people believe that they do not get enough iron and that TE is the result. Other potential shortcomings of the modern North American diet such as zinc, amino acid L-lysine or vitamin B6 and B12 deficiency have also been suggested to contribute to TE.
When dietary deficiencies are suspected, reinforcements may be available. However, the reinforcements themselves can cause problems. Our bodies can only handle too much iron each day. High doses of iron toxicity can cause hair loss. In really high doses, iron supplements cause death. Excessive vitamin A may also be toxic, some vitamin A supplements may cause the TE reaction.
TE may appear on its own or as part of another disease. The early stages of androgenetic alopecia (male or female pattern baldness, briefly AGA) are effectively TE. Early AGA is characterized by an increase in telogen hair follicles at rest. One of the earliest stages of AGA is telangiectasia of scalp hair follicles up to 40%.
TE can also be a symptom of other conditions, such as inflammatory conditions such as alopecia areata. Hair follicles are particularly susceptible to thyroid hormones and about three times of individuals with thyroid disorders have TE. Exposure to toxins can also cause TE as one of the many symptoms.